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Chapter 8-1. DNA Correction and Repair

Recommended Reading: 【Biology】 Chapter 8. Central Dogma


1. Mutagens

2. Original Mark

3. Correction

4. Repair

5. Failure of DNA Correction and Repair



1. Mutagens



2. Original Mark

⑴ Methylates the A base of the template strand to mark it as the original.

⑵ Dna methylase is involved.

⑶ Methylates the 5’-GATC-3’ sequence in the ori C region.



3. Correction

⑴ DNA polymerase has correction functions, and the replication error rate is 1/107.

Mechanism 1. Exonuclease

Mechanism 2. 3’ → 5’ correction (DNA pol I, II, III): The 3’ → 5’ exonuclease activity allows correction of errors that occur during DNA synthesis.

Mechanism 3. 5’ → 3’ correction (DNA pol I): 5’ → 3’ exonuclease activity



4. Repair

⑴ If repair enzymes correct errors, the replication error rate is 1/109.

Type 1. Dimer recovery


image

Figure 1. Dimer recovery mechanism


① 1st. Ultraviolet light induces pyrimidine dimers (TT) between adjacent thymines.

② 2nd. DNA photolyase recognizes changes in the phosphodiester backbone and binds to the pyrimidine dimer.

③ 3rd. DNA photolyase absorbs blue light (visible light over 300 nm) and becomes activated.

④ 4th. The activated DNA photolyase separates thymine from the DNA.

Type 2. Excision Repair

① Overview

○ The most common repair system capable of fixing various structural damages in DNA.

○ Best characterized in E. coli, with various modified repair mechanisms present in other organisms.

Type 2-1. Mismatch excision repair

○ Characteristics: Uses original marks, Mut proteins, and DNA pol III.


image

Figure 2. Excision repair process in E. coli


Type 2-2. Base excision repair

○ Definition: A repair method for cases where bases are damaged by oxidation, alkylation, hydrolysis, or deamination, forming U bases.

Process 1. DNA glycosylase cleaves between the damaged base and the pentose sugar, removing only the base.

Process 2. AP endonuclease cleaves the sugar-phosphate backbone at the 5’ side of the AP site where only the base has been removed.

○ AP: apurine or apyrimidine

Process 3. AP lyase, endonuclease, phosphodiesterase: Remove the remaining parts of the damaged nucleotide.

Process 4. DNA polymerase: Synthesizes nucleotides in the removed region.

○ In E. coli, DNA pol I is involved, while in mammals, DNA pol β is involved.

Process 5. DNA ligase: Seals the nick.

Type 2-3. Nucleotide excision repair

○ Definition: A repair process for large, helix-distorting damages such as pyrimidine dimers.

Process 1. Incision step

Step 1-1. Repair endonuclease: Recognizes and cleaves the distorted region caused by thymine dimers.

Step 1-2. Cuts the sugar-phosphate bond 8 nucleotides upstream of the thymine dimer on the 5’ side.

Step 1-3. Cleaves the sugar-phosphate bond 4–5 nucleotides downstream of the thymine dimer on the 3’ side.

○ Ultimately, two cuts are made 12–13 nucleotides apart.

○ Helicase and exonuclease are involved in steps 1-2 and 1-3.

Process 2. DNA polymerase I (pol I) binds to the 3’-OH group at the end of the cut site.

Step 2-1. Removes the DNA fragment containing the thymine dimer while synthesizing a new strand.

Step 2-2. In eukaryotes, DNA pol ε/δ is involved.

Process 3. Ligase seals the newly synthesized fragment into the original DNA strand.

Type 3. High-fidelity post-replication repair: Increased Lex A protein → Increased accuracy

Type 4. SOS repair: Emergency repair mechanism. Decreased Lex A protein → Increased Rec A protein activity → Decreased accuracy



5. Failure of DNA Correction and Repair

⑴ Leads to frequent mutations.



Input: 2019.03.03 00:37

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